V900884
十二烷基三甲基氯化铵
98%, Vetec™, reagent grade
别名:
N-Dodecyl-N,N,N-trimethylammonium chloride, DTAC, Trimethyldodecylammonium chloride
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线性分子式:
CH3(CH2)11N(CH3)3Cl
化学文摘社编号:
分子量:
263.89
UNSPSC Code:
12352107
PubChem Substance ID:
EC Number:
203-927-0
Beilstein/REAXYS Number:
3915951
MDL number:
Assay:
99.1%, ≥98%
Form:
solid
产品名称
十二烷基三甲基氯化铵, Vetec™, reagent grade, ≥98%
grade
reagent grade
product line
Vetec™
assay
99.1%, ≥98%
form
solid
reaction suitability
core: ammonium
mp
37 °C
functional group
amine
SMILES string
[Cl-].CCCCCCCCCCCC[N+](C)(C)C
InChI
1S/C15H34N.ClH/c1-5-6-7-8-9-10-11-12-13-14-15-16(2,3)4;/h5-15H2,1-4H3;1H/q+1;/p-1
InChI key
DDXLVDQZPFLQMZ-UHFFFAOYSA-M
General description
十二烷基三甲基氯化铵(DTAC)是一种季铵化合物(QAC)，常用作阳离子表面活性剂。主要用于蛋白质变性。
Application
DTAC可用作下列反应中的阳离子表面活性剂：
- 酮/醛的转移氢化反应。
- 烯烃的开环复分解聚合反应。
Legal Information
Vetec is a trademark of Merck KGaA, Darmstadt, Germany
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signalword
Warning
hcodes
Hazard Classifications
Acute Tox. 4 Oral - Aquatic Acute 1 - Eye Irrit. 2 - Skin Irrit. 2
存储类别
13 - Non Combustible Solids
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
Johannes Weirather et al.
Circulation research, 115(1), 55-67 (2014-05-03)
An exaggerated or persistent inflammatory activation after myocardial infarction (MI) leads to maladaptive healing and subsequent remodeling of the left ventricle. Foxp3(+) CD4(+) regulatory T cells (Treg cells) contribute to inflammation resolution. Therefore, Treg cells might influence cardiac healing post-MI.
Jing Shang et al.
Scientific reports, 5, 13651-13651 (2015-09-04)
In-cell immunoassays have become a valuable tool for protein expression analysis complementary to established assay formats. However, comprehensive molecular characterization of individual specimens has proven challenging and impractical due to, in part, a singleplex nature of reporter enzymes and technical
Sara G Hovakeemian et al.
Soft matter, 11(34), 6840-6851 (2015-08-04)
Most antimicrobial peptides act upon target microorganisms by permeabilizing their membranes. The mode of action is often assessed by vesicle leakage experiments that use model membranes, with the assumption that biological activity correlates with the permeabilization of the lipid bilayer.